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. Author manuscript; available in PMC: 2010 Nov 19.
Published in final edited form as: Circ J. 2010 Jan 18;74(3):390–404. doi: 10.1253/circj.cj-09-0923

Figure 5.

Figure 5

Cardiac function assessed by echocardiography and hemodynamics in rats after myocardial infarction (MI). (A) Quantitative echocardiographic parameters including systolic thickness of the anterior (infarcted) wall, thickening fraction in the anterior (infarcted) wall, left ventricular (LV) fractional shortening, and LV ejection fraction in vehicle-treated (n=14) and cardiac progenitor cell (CPC)-treated group (n=17) at baseline (BSL), at 30 days (d) after MI (before vehicle or CPC treatment) (MI), and at 35 d after treatment. (B) Quantitative analysis of hemodynamic variables including LV end-diastolic pressure, dP/dt, ejection fraction, end-systolic elastance (Ees, preload-adjusted maximal power, and preload-recruitable stroke work from normal (n = 4), vehicle-treated (n=15), and CPC-treated rats (n=15) at 35 d after treatment. Data are means±SEM. (Reprinted from Tang et al.44)