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. 2010 Aug 31;53(8):779–785. doi: 10.3345/kjp.2010.53.8.779

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Copyright © 2010 by The Korean Pediatric Society

This is an open-access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 1 — Convergent multimodal imaging in a 11-year-old boy with left temporal lobe epilepsy. Cortex thickening in the left temporal lobe is seen on the corresponding coronal T1W1 MRI. SISCOM demonstrated a cluster of hyperperfusion in the left temporal lobe, consistent with the patient's ictal EEG. 18-FDG PET shows reduced metabolism in the overlying cortex. All imaging data were concordant with electroclinical and neuropsychological data and the patient underwent a neocortical temporal lobectomy. The patient has since remained seizure-free. Histopathology after temporal lobectomy showed FCD type 2A.