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. Author manuscript; available in PMC: 2010 Dec 21.
Published in final edited form as: Biochem Biophys Res Commun. 2009 Jun 6;386(1):101–104. doi: 10.1016/j.bbrc.2009.05.132

Fig. 4.

Fig. 4

(A) Activation of the JAK-STAT pathway and the Ras-MAPK pathway in NECs treated with tunicamycin. Lysates prepared from NECs treated with tunicamycin (0 or 5 μg/ml) for 8 h and then stimulated with LIF (0 or 40 ng/ml) for 10 min were analyzed by Western-blot analysis. Stress-mediated cell death signaling was evaluated by measuring (B) LDH activities in culture media of tunicamycin-treated NECs and (C) caspase 3 activities in tunicamycin-treated NECs.