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. 2010 Dec 15;7:92. doi: 10.1186/1742-2094-7-92

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Copyright ©2010 Bao et al; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (<url>http://creativecommons.org/licenses/by/2.0</url>), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Effect of MFX on post-ischemic inflammation and injury in the brain. MCP-1 mRNA (A) and CCR2 mRNA (B) levels were measured in the 7 d post-ischemic brain in vehicle (Veh) and MFX treated mice. CCR2 mRNA level (B) was decreased by MFX, and MCP-1 was not significant between Veh and MFX treatment (A). β-actin was used as internal control and data were normalized with the contralateral side in Veh group. Values were expressed as mean ± SEM. Contl, contralateral side; Ipsl, ipsilateral side. ** p < 0.01 and *** p < 0.001 compared to contralateral side; # p < 0.05 compared to Veh; NS, not significant. n = 6-8/group. C, Infarct volume measurement 7 d after MCAO. Values were present as mean ± 95% Confidence interval. * p < 0.05 compared to Veh. n = 12-15/group.