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. Author manuscript; available in PMC: 2011 Jan 11.
Published in final edited form as: Arch Ophthalmol. 2007 Feb;125(2):239–245. doi: 10.1001/archopht.125.2.239

Figure 1.

Figure 1

Prevalences of retinal angiomatosis in VHL patients in 3 genotypic mutational categories of single amino-acid substitutions, protein-truncating mutations, and complete deletion of VHL protein. Asterisk (*) indicates that the prevalence for complete deletions were significantly lower than those for amino-acid substitutions and protein truncations (p<0.0001).