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. Author manuscript; available in PMC: 2011 Jan 12.
Published in final edited form as: Nat Immunol. 2006 Dec 3;8(1):92–100. doi: 10.1038/ni1414

Table 1.

Increase in hydrogen-bonding distance after His → Asn

PDB ID Allele–peptide residue d(His) d(Asn) (PyMol) Change
1DLH DR1–Lys2 2.69Å 3.86Å (3.75) 1.17Å
1A6A DR3–Lys2 3.03Å 3.80Å (3.75) 0.77Å
1AQD DR1–Asp4 2.68Å 3.67Å (3.48) 0.99Å
1H15 DR2–Val3 2.77Å 3.85Å (3.74) 1.07Å
1HQR DR2–Phe3 3.27Å 4.14Å (4.10) 1.17Å
1HXY DR1–Lys2 2.66Å 3.73Å (3.66) 1.08Å
1J8H DR1–Lys2 2.57Å 3.65Å (3.55) 1.08Å
1KLU DR1–Leu3 3.00Å 4.17Å (4.13) 1.17Å
1KT2 I-Ek–Leu4 3.03Å 4.20Å (4.02) 1.17Å
1KTD I-Ek–Leu4 2.88Å 4.04Å (3.86) 1.16Å
1IEA I-Ek–Val4 2.72Å 4.11Å (3.98) 1.39Å

Modeling of His→Asn substitution with Swiss PDBviewer or PyMol41 for various human and mouse MHC class II molecule crystal structures. All structures have the conserved histidine residue; however, the side chains of the peptides whose carbonyl group potentially form a hydrogen bond with that histidine residue vary (column 2). d(His), distance between the conserved histidine and the oxygen atom of carbonyl group, as in the structure; d(Asn), distance between closest allowed 'conformer' of asparagine (after the substitution) and that same carbonyl group; change, d(Asn) – d(His). The potential hydrogen-bonding distance consistently increases by an average of 1.1Å; hence, the bond is weaker for the asparagine mutant models for all structures analyzed. PDB ID, Protein Data Bank accession number.