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. 2011 Jan 20;6(1):e16340. doi: 10.1371/journal.pone.0016340

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Palande et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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ChIP using αH3K27me3 and an IgG control (A) and αH3K4me3 and an IgG control (B) showing an enrichment of both H3K27me3 and H3K4me3 at the TSS of PRDX4 in APL cells. To rule out a possible involvement of X-inactivation, and thereby H3K27me3, in silencing of PRDX4 expression, male APL samples are compared to a male AML control and female APL samples are compared to a female AML control.