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. 2011 Jan 21;60(2):391–397. doi: 10.2337/db10-0426

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© 2011 by the American Diabetes Association.

Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.

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FIG. 1. — A: Comparison of plasma glucagon levels in Gcgr^+/+ (■) and Gcgr^−/− (□) mice before and 6 weeks after STZ induction of β-cell destruction. B: Glucagon in Gcgr^+/+ mice before and after STZ. C: Representative immunoblots for CREB and P-CREB in livers of Gcgr^+/+ (■) and Gcgr^−/− (□) mice after β-cell destruction by treatment with STZ and in untreated controls (upper panel). The P-CREB/CREB ratio in densitometric units in six Gcgr^+/+ (■) and six Gcgr^−/− (□) mice after β-cell destruction by treatment with STZ (lower panel).