Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2011 Jan 21;60(2):486–495. doi: 10.2337/db10-0650

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2011 by the American Diabetes Association.

Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.

PMC Copyright notice

FIG. 7. — Adipose tissue–specific inactivation of JNK alleviated the HFD-induced inflammatory changes. A: Quantitative RT PCR analysis for the gene expression of F4/80 and the inflammatory cytokines MCP-1, TNF-α, and IL-6 in epididymal fat. Expression levels of all genes were normalized against 18S rRNA. B: Immunohistochemical staining for F4/80 in epididymal fat tissue sections from aP2-dn-JNK transgenic (Tg) mice and wild-type (WT) littermates. Scale bar: 50 μm. C: Serum leptin, adiponectin, and A-FABP levels in overnight-fasted aP2-dn-JNK transgenic mice and wild-type littermates were measured by an enzyme-linked immunosorbent assay after being fed an HFD for 16 and 20 weeks, respectively. □, wild-type littermates; ■, aP2-dn-JNK transgenic mice. *P < 0.05; **P < 0.01 vs. wild-type littermates (n = 6–8). (A high-quality color representation of this figure is available in the online issue.)