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. 2010 Dec 3;193(4):842–853. doi: 10.1128/JB.01004-10

FIG. 4.

FIG. 4.

Effect of σE activation on ompR malT(Con) survival. (A) Colony phenotypes of the ompR malT(Con) mutant (AJW3098) (left) and the ompR malT(Con) rseA::Tn5 mutant (strain AJW3855) (right). Colonies were grown in LB at 37°C. (B) Growth curves of WT (AJW678) (closed circles), ompR malT(Con) (AJW3098) (closed diamonds), rseA (AJW3815) (open circles), and ompR malT(Con) rseA (AJW3818) (open diamonds) strains. Cells were grown in LB at 37°C. Values represent the means of triplicates, and error bars indicate standard deviations and are shown only when greater than the size of the symbol. (C) β-Galactosidase activities of WT (AJW678), ompR malT(Con) (AJW3098), rseA (AJW3815), and ompR malT(Con) rseA (AJW3818) strains carrying a Φ(rpoHP3-lacZ) promoter fusion grown under nonpermissive conditions. Cells were harvested at an OD600 of 1. Values represent the means and standard deviations for triplicates.