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. 2010 Aug 21;131(2):275–281. doi: 10.1007/s00402-010-1171-7

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© The Author(s) 2010

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Fig. 1 — Comparison of the frequency rate of TLR4 gene mutation in positions Asp299Gly (left) and Thr399Ile (right) marked as 1/W and 2/W, respectively, a in patients with delayed fracture healing and viable bacteria isolated from fracture gaps (darkly shaded bar) and patients with delayed healing and sterile fracture gaps (lightly shaded bar). W wild allele; 1/W, 2/W mutations (%), Chi-square test, *P < 0.05, b with delayed healing and bacteria isolated from fracture (darkly shaded bar) and patients with uneventful healing fractures also with bacteria (lightly shaded bar) (NS), c with delayed healing and sterile gaps (darkly shaded bar) and patients with uneventful healing fractures also with sterile gaps (lightly shaded bar) (NS), d with uneventful healing and bacteria isolated from fracture (darkly shaded bar) and uneventful healing fractures also without bacteria (lightly shaded bar) (NS)