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. 2011 Jan 20;2011:183256. doi: 10.4061/2011/183256

Table 5.

Investigations.

Young patients (n = 100) Old patients (n = 1117) P
ECG on admission
Total 82 (82) 1057 (94.6)
Atrial fibrillation 2 (2.4) 181 (17.1) <.001
Left bundle branch block 0 (0) 38 (3.6) .11
Left ventricle hypertrophy 6 (7.3) 73 (6.9) .82
Unspecific ST depression 7 (8.5) 232 (21.9) .003
Acute anterior myocardial infarction 0 (0) 3 (.3) 1.00
Old anterior myocardial infarction 2 (2.4) 52 (4.9) .42
Acute inferior myocardial infarction 0 (0) 2 (.2) 1.00
Old inferior myocardial infarction 2 (2.4) 59 (5.6) .31

Echocardiography
Total 63 (63) 357 (32.0)
TTE 28 (44.4) 284 (79.6)
TEE 35 (55.6) 73 (20.4)
Left ventricle hypertrophy 7 (11.1) 119 (33.3) <.001
Patent foramen ovale 10 (15.9) 14 (3.9) .001
Sequelae anterior myocardial infarction 2 (3.2) 19 (5.3) .75
Sequelae inferior myocardial infarction 0 (0) 16 (4.5) .15

Holter monitoring
Total 57 (57) 434 (38.9)
Paroxysmal atrial fibrillation 1 (1.8) 78 (18.0) .001

Duplex of cervical arteries
Total 86 (86) 893 (79.9)
ICA stenosis1 11 (12.8) 356 (39.9) .000
Symptomatic ICA stenosis ≤49%1* 0 (0) 83 (13.9) .002
Symptomatic ICA stenosis 50–69%1* 0 (0) 55 (9.2)
Symptomatic ICA stenosis 70%–99%1* 2 (3.9) 34 (5.7)
Symptomatic occlusion1* 5 (9.8) 29 (4.9)
No ICA stenosis1* 44 (86.3) 397 (66.4)

Data are expressed as mean or n (%).

ECG, electrocardiography; ICA, internal carotid artery.

¹Area reduction measured by neurosonology.

*Among patients with ipsilateral infarction in the middle cerebral artery territory.