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. 2010 Jun;51(6):1344–1353. doi: 10.1194/jlr.M002196

Fig. 3.

Fig. 3.

Expression of genes that control fatty acid levels in liver after CSDS. (A) Significant differences in expression of genes involved in fatty acid synthesis: FAS, SCD1, ACC1a, SREBP1c, and LXRα. (B) Expression of genes that regulate fatty acid utilization: PDK4, UCP2, FGF21, CPT1a, PPARα, and CD36. Data are presented as mean ± SEM, with significant differences as an insert on the chart (P < 0.05, P < 0.01, P < 0.001). Groups tested by two-way ANOVA. ACC1a, acetyl CoA carboxylase 1a; CPT1a, carnitine palmitoyltransferase 1a; CSDS, chronic social defeat stress; FGF21, fibroblast growth factor 21; LXRα, liver X receptor α; PDK4, pyruvate dehydrogenase kinase 4; PPARα, peroxisome proliferator-activated receptor α; SCD1, stearoyl CoA desaturase 1; SREBP1c, sterol-regulatory element binding protein 1c; UCP2, uncoupling protein 2.