Skip to main content
. 2011 Mar 15;138(6):1183–1193. doi: 10.1242/dev.060905

Fig. 1.

Fig. 1.

daf-28 acts with ins-6 to inhibit dauer entry, whereas ins-1 promotes it. (A,B) The mean fractions of wild type and insulin-deletion C. elegans mutants that form dauers at 25°C (A) or 27°C (B). Each mean ± s.e.m. includes at least three independent trials of ~100 worms per trial. The detailed statistical comparisons between the dauer entry phenotypes of different genotypes under different conditions in this and subsequent figures can be found in Table S1 in the supplementary material. (C,D) The effect of different insulin deletions on the dauer entry of daf-2(e1368) mutants at 22.5°C (C) and 20°C (D). (E) The dauer entry of ins-6; daf-28 deletion mutants is suppressed by daf-16(mu86) at 25°C. *, P≤0.05; **, P≤0.01; ***, P≤0.001.