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. Author manuscript; available in PMC: 2012 Aug 1.
Published in final edited form as: Biochim Biophys Acta. 2010 Oct 20;1812(8):893–908. doi: 10.1016/j.bbadis.2010.10.006

Figure 4. Integrated view of SHP as a central transcriptional coregulator in diverse physiological pathways in the liver.

Figure 4

Activation pathways are shown as broken blue arrows and inhibitory pathways are shown as broken red lines. FA, fatty acid; TG, triglyceride; VLDL, very low density lipoprotein; CYP7A1, cytochrome P450 enzyme cholesterol 7α-hydroxylase; G6P, glucose-6-phosphatase; HNF-4α, hepatocyte nuclear factor 4 α; JNK, c-jun N-terminal kinase; LRH-1, liver receptor homolog-1; LXR, liver X receptor; MTP, microsomal triglyceride transfer protein; PEPCK, phosphoenolpyruvate carboxykinase; PPARγ, peroxisome proliferator activated receptor γ; SHP, small heterodimer partner; SREBP-1c, sterol regulatory element binding protein-1c; WAT, white adipose tissue; and BAT, brown adipose tissue.