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. Author manuscript; available in PMC: 2012 Jan 1.
Published in final edited form as: Ann Neurol. 2011 Jan;69(1):181–192. doi: 10.1002/ana.22248

FIGURE 3.

FIGURE 3

Box and whiskers plots showing the relation between the number of apolipoprotein E (ApoE) ε4 alleles and change in neocortical [11C]Pittsburgh compound B (PiB) standardized uptake value ratio (SUVR) at 20-month follow-up. ApoE ε4 heterozygotes (HTZ) and homozygotes (HMZ) had a significant gene dose-dependant increase in neocortical PiB SUVR at follow-up when compared to noncarriers in the mild cognitive impairment group, but not in health controls and dementia of the Alzheimer type. †p < 0.05.