. 2011 Feb 25;6(2):e17117. doi: 10.1371/journal.pone.0017117

Table 2. Effects of aminoguanidine, TEA, losartan and enalapril on the vascular responses to phenylephrine (R_max and pD₂) in aortas from untreated and lead-treated rats.

	Untreated		Lead treated
	Rmax	pD2	Rmax	pD2
Control	82.06±6,48	6.02±0,15	60.35±5,99^#	6.14±0.08
Aminoguanidine	86.93±5.56	6.36±0,19	83.71±2,57*	6.23±0.08
TEA	100.04±3.70^#	6.50±0,11^#	98.29±5,30*	6.30±0,08
Enalapril	79.12±2.69	6.21±0.13	48.93±2.53*	6.20±0.13
Losartan	79.77±4.12	6.19±0.05	35.13±5.89*	5.98±0.12

Results are expressed as mean ± SEM of the number of animals shown in Figs. 3 and 5 ; R_max, maximal effect (expressed as a percentage of the maximal response induced by 75 mM KCl); pD₂, −log one-half R_max; AG; aminoguanidine, TEA; tetraethylammonium, losartan, enalapril. P<0.05 vs. untreated control rats (^#) and lead-treated control rats (*).

Table 2. Effects of aminoguanidine, TEA, losartan and enalapril on the vascular responses to phenylephrine (Rmax and pD2) in aortas from untreated and lead-treated rats.

Table 2. Effects of aminoguanidine, TEA, losartan and enalapril on the vascular responses to phenylephrine (R_max and pD₂) in aortas from untreated and lead-treated rats.