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. 2011 Mar;4(2):268–276. doi: 10.1242/dmm.002998

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© 2011. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Share Alike License (http://creativecommons.org/licenses/by-nc-sa/3.0), which permits unrestricted non-commercial use, distribution and reproduction in any medium provided that the original work is properly cited and all further distributions of the work or adaptation are subject to the same Creative Commons License terms

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Fig. 5. — SeAP activity falls in the serum of adult BAC NEUROG3-SeAP/EGFP transgenic mice after partial pancreatectomy but increases after pancreatic duct ligation. Day 0 is prior to surgery. (A) The white bar at day 0 shows the background signal for SeAP activity in the serum of non-transgenic mice. Gray bars show the activity of SeAP in the serum of transgenic animals that underwent a sham operation. Black bars show the activity of SeAP in the serum of transgenic animals that underwent partial pancreatectomy. Data represent mean + s.e.m. from three mice in each treated group, and four mice in the untreated, non-transgenic group. ***P<0.001 compared with serum from sham-operated mice by Student’s t-test. (B) The white bars show the background signal for SeAP activity in the serum of control transgenic mice that did not have surgery. Gray bars show the activity of SeAP in the serum of animals that underwent a sham operation. Black bars show the activity of SeAP in the serum of animals that underwent duct ligation. Data represent mean + s.e.m. from six mice in the untreated group, and seven mice in each treated group. *P<0.03, **P<0.01, ***P<0.001 by Student’s t-test for the comparisons indicated.