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. 2011 Feb 21;60(3):947–959. doi: 10.2337/db10-1145

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© 2011 by the American Diabetes Association.

Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.

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FIG. 3. — A, left side: Representative line-scan images of a single electrically evoked global Ca²⁺ transient and contraction kinetics (right side) of ventricular myocyte from control (C) and STZ-induced diabetic (D) rats. Values shown in the lower panel are means ± SE for n > 50 cells. *Significantly different from control rats (P < 0.05). B, left side: Consecutive evoked Ca²⁺ transients (≥20) in ventricular myocytes isolated from control (C), STZ-induced diabetic (D), and insulin-treated STZ-induced diabetic (Ins-D) rat hearts stimulated at 2 Hz. B, right side: Consecutive evoked contractions (≥20) of ventricular myocytes isolated from control (C), STZ-induced diabetic (D), and insulin-treated STZ-induced diabetic (Ins-D) rat hearts stimulated at 2 Hz. Black arrows indicate application of field stimulation (2 Hz), and red lines indicate abnormal Ca²⁺ transients and contractions. White arrows (left panel) represent variation in Ca²⁺ decay time along the scanned segment. (A high-quality digital representation of this figure is available in the online issue.)