Skip to main content
. 2011 Mar;163(3):368–374. doi: 10.1111/j.1365-2249.2010.04303.x

Table 1.

Clinical features of proteolipid protein (PLP)-induced experimental allergic encephalomyelitis (EAE) in Swiss Jackson Laboratory (SJL) mice upon treatment with carbon monoxide-releasing molecule (CORM)-A1, dexamethasone or vehicle.

Experimental groups Cumulative score 0–30 Cumulative score 31–45 Onset Duration Incidence % Incidence after interr. (%)
CORM-A1 early prophylaxis (0–30) 4·3 ± 7·3 2·8 ± 7·8 16·7 ± 1·5 2 ± 3·2 37·5 37·5
CORM-A1 prophylaxis (0–16) 15·3 ± 11·2 22·9 ± 4·6 10·9 ± 6·5 88·9
CORM-A1 late prophylaxis (10–30) 4·8 ± 9·4 20·7 ± 5·5 2·2 ± 4·2 30·0
Dexamethasone 6 ± 6·9 11·4 ± 11·6 18·4 ± 4·3 3·3 ± 3·2 62·5 75·0
Vehicle 19·6 ± 16 17·4 ± 14·1 17·5 ± 3·9 8·3 ± 6·0 85·7 85·7
Mann–Whitney
CORM-A1 early prophylaxis (0–30) 0·04 0·03 0·90 0·03
CORM-A1 prophylaxis (0–16) 0·94 0·71 0·31
CORM-A1 late prophylaxis (10–30) 0·05 0·36 0·05
Dexamethasone 0·07 0·34 0·65 0·06