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. 2011 Jan 25;104(4):664–672. doi: 10.1038/sj.bjc.6606062

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Copyright © 2011 Cancer Research UK

This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material.If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit https://creativecommons.org/licenses/by/4.0/.

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Extracellular signal-regulated protein kinase 5 protein and mir143 transcript expression in clinical prostate cancer (PCa) by immunohistochemistry (IHC) and in situ hybridisation (ISH) respectively. Extracellular signal-regulated protein kinase 5 immunoreactivity in (A) Gleason 7 PCa, (B) Gleason 9 PCa, (C) BPH, (D) normal prostate, (E) PIN, (F) Castrate-resistant PCa (CRPC), (G) liver metastasis, (H) lymph node metastasis; (I and J) mir143 mRNA and ERK5 protein expression analysis in sequential slides from two cases of PCa (solid and open arrows signify ERK5 protein and mir143 transcript expression, respectively), (K) mir143 expression in endothelium; β-actin expression in (L) BPH and (M) PCa (scale bar represents 100 μm).