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. Author manuscript; available in PMC: 2011 Apr 1.
Published in final edited form as: Alcohol Clin Exp Res. 2010 Jan 26;34(4):594–606. doi: 10.1111/j.1530-0277.2009.01128.x

Figure 1.

Figure 1

AMPAR- and NMDAR-mediated fEPSP input-output curves in the CA1 hippocampus. Sample traces of (A) AMPAR- and (B) NMDAR-mediated fEPSPs evoked at increasing stimulus intensities via stimulation of the Schaffer collateral fibers. Events were recorded in the stratum radiatum in the presence of gabazine (10 μM) and AP5 (50 μM) for AMPARs, and in the presence of gabazine (10 μM), NBQX (10 μM), and absence of Mg2+ for NMDARs. Pooled data for (C) AMPAR- and (D) NMDAR-mediated fEPSP input-output curves. (E) AMPAR- and (F) NMDAR-mediated fEPSPs were blocked with GYKI-53655 (50 μM) and AP5 (50 μM), respectively.