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. 2011 Mar 7;192(5):873–882. doi: 10.1083/jcb.201009132

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© 2011 Choi et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 5. — Oxidative stress contributes to rotenone-induced dopamine neuron death. (A) 5 nM rotenone for 12 h increases ROS production in TH⁺ neurons, which is prevented by cotreatment with 10 nM taxol. (B) Cotreatment with 0.5 mM N-acetylcysteine (NAC), an antioxidant, reduces 24-h 5-nM rotenone–induced TH⁺ neuron death. Data are means ± SEM. n = 3; *, P < 0.05; **, P < 0.01; ***, P < 0.001.