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. Author manuscript; available in PMC: 2011 Mar 11.
Published in final edited form as: Cell Metab. 2011 Mar 2;13(3):308–319. doi: 10.1016/j.cmet.2011.02.002

Figure 3. Oral GTT after in humans with inactivating PRKAR1A mutations and controls.

Figure 3

A) Subjects with a PRKAR1A mutation exhibit normal fasting glucose levels but reduced glucose excursion after an oral glucose load.

B) In subjects with a PRKAR1A mutation serum insulin levels were not different at baseline, and reached a higher peak with increased overall insulin secretion.

C) Table summarizing fasting glucose and insulin levels as well as area under the glucose and insulin curves shown in A and B (*p<0.05)