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. Author manuscript; available in PMC: 2012 Mar 1.
Published in final edited form as: Atherosclerosis. 2010 Dec 22;215(1):52–59. doi: 10.1016/j.atherosclerosis.2010.12.009

Figure 3. Lipid accumulation in the innominate arteries following P. gingivalis infection.

Figure 3

Representative images of birefringence in plaque corresponding to lipids (total cholesterol esters and cholesterol monohydrate crystals) are shown. (A) group i: uninfected / non-immunized, (B) group ii: uninfected / immunized, (C) group iii: P. gingivalis infected / non-immunized, and (D) group iv: P. gingivalis infected / immunized ApoE−/− mouse. Bar represents 200 μm. (E) Lipid area. In non-immunized ApoE−/− mice P. gingivalis infection increased lipid area compared to uninfected ApoE−/− mice. In P. gingivalis infected ApoE−/− mice, the lipid area was significantly decreased in the immunized group compared to non-immunized group. *p < 0.01, One-way ANOVA. NS indicates no significant difference.