Skip to main content
. Author manuscript; available in PMC: 2012 Jan 1.
Published in final edited form as: Microcirculation. 2011 Jan;18(1):36–45. doi: 10.1111/j.1549-8719.2010.00066.x

Figure 4.

Figure 4

NO production by coronary arterioles in the presence and absence of OxLDL. NO production was measured by chemiluminescence detection of nitrite. The resting NO production (Basal; n=6) was not altered by OxLDL (0.5 mg protein/ml; n=6). In the presence of OxLDL (0.5 mg protein/ml; n=4), the NO production stimulated by serotonin (0.1 μM; n=4) was significantly inhibited. In the presence of arginase inhibitor DFMO (0.4 mM), the serotonin-induced NO production (0.1 μM; n=5) was comparable to that without DFMO and the OxLDL (0.5 mg protein/ml; n=5) did not affect this NO production. *P<0.05 vs. Basal.