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. Author manuscript; available in PMC: 2012 Apr 1.
Published in final edited form as: Biochim Biophys Acta. 2010 Dec 28;1815(2):197–213. doi: 10.1016/j.bbcan.2010.12.002

Fig. 2.

Fig. 2

Potential Notch signaling crosstalk with other pathways in breast cancer. OB-R: leptin receptor ; IL-1: pro-inflammatory/-angiogenic cytokine interleukin-1; NILCO: Notch-IL-1-Leptin crosstalk outcome; E2: 17β-estradiol; HER/ErbB: HER1/EGFR, HER2, HER3 and HER4, encode for RTK-transmembrane proteins; NF-κB: a transcription factor family, nuclear factor kappa-light-chain-enhancer of activated B cells; IL-6: pro-inflammatory/-angiogenic cytokine, interleukin-6; STAT3: Signal transducer and activator of transcription 3; PDGF-D: Platelet-derived growth factor D; TGF-β: transforming growth factor β; miRNA: MicroRNA; PI-3K: phosphatidylinositol 3-kinase; mTOR: mammalian target of rapamycin; HIF-1: hypoxia-inducible transcription factor 1; VEGF: vascular endothelial growth factor; VEGFR-2: vascular endothelial growth factor receptor-2.