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. Author manuscript; available in PMC: 2011 Sep 1.
Published in final edited form as: J Thromb Haemost. 2011 Mar;9(3):552–561. doi: 10.1111/j.1538-7836.2010.04160.x

Figure 1.

Figure 1

Platelet aggregations induced by ADP (0.1-30 μmol L−1; panel A), collagen (0.1-30 μg mL−1; panel B), or U46619 (0.1-30 μmol L−1; panel C) were inhibited in a concentration-dependent manner by PAM (p<0.05 for all; two way ANOVA). Curves for 0.1 μmol L−1 and 0.3 μmol L−1 ADP are superimposed upon one another (panel A), as are curves for 0.1 μmol L−1 and 0.3 μmol L−1 U46619 (panel B). Data shown are mean ± s.e.m. of responses measured by 96-well plate aggregometry in citrated PRP prepared from 6 different individuals.