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. 2010 Jul 12;15(6):1319–1328. doi: 10.1111/j.1582-4934.2010.01126.x

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© 2011 The Authors Journal of Cellular and Molecular Medicine © 2011 Foundation for Cellular and Molecular Medicine/Blackwell Publishing Ltd

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Fig 4 — Impact of cell therapy on LV function. (A)–(C) Representative M-mode images at 35 days after coronary occlusion/reperfusion from mice that were given vehicle (group I) (A), expanded and untreated VSEL-SCs (group II) (B) and expanded pre-incubated VSEL-SCs (group III) (C). Compared with the hearts in groups I and II, the heart in group III exhibited a smaller LV cavity, a thicker infarct wall and improved motion of the infarct wall. (D–E) Quantitative echocardiographic analysis revealed improvement in LV functional parameters following transplantation of pre-incubated VSEL-SCs at 35 days after MI. Data are means ± S.E.M. n= 7–11 mice/group. *P<0.05 versus group I at 35 days.

Fig 4 — Impact of cell therapy on LV function. (A)–(C) Representative M-mode images at 35 days after coronary occlusion/reperfusion from mice that were given vehicle (group I) (A), expanded and untreated VSEL-SCs (group II) (B) and expanded pre-incubated VSEL-SCs (group III) (C). Compared with the hearts in groups I and II, the heart in group III exhibited a smaller LV cavity, a thicker infarct wall and improved motion of the infarct wall. (D–E) Quantitative echocardiographic analysis revealed improvement in LV functional parameters following transplantation of pre-incubated VSEL-SCs at 35 days after MI. Data are means ± S.E.M. n= 7–11 mice/group. *P<0.05 versus group I at 35 days.