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. Author manuscript; available in PMC: 2011 Dec 1.
Published in final edited form as: Arterioscler Thromb Vasc Biol. 2010 Sep 23;30(12):2518–2527. doi: 10.1161/ATVBAHA.110.215350

Figure 1.

Figure 1

Glucose homeostasis and systemic inflammation in wild-type C57BL/6 mice and p47phox−/− mice by ambient PM2.5 exposure compared with FA-exposed mice fed an ND (n = 16) or an HFD (n = 16). A and E, Effect of PM2.5 exposure on glucose tolerance by intra-peritoneal glucose tolerance test (IPGTT) in C57BL/6 mice and in p47phox−/− mice, respectively. B and F, The glucose area under the curve calculated from the glucose tolerance test from parts A and E, respectively. C and G, The homeostasis model assessment IR index in C57BL/6 mice and in p47phox−/− mice, respectively. D and H, Plasma cytokine measurement by ELISA in C57BL/6 mice and in p47phox−/− mice, respectively. n = 8 in each group. *P<0.05 and **P<0.001. IFN indicates interferon; MCP-1, monocyte chemoattractant protein-1; RANTES, regulated on activation, normal T cell expressed and secreted (or chemokine C-C motif ligand 5).