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. 2011 Apr;28(4):547–563. doi: 10.1089/neu.2010.1729

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Copyright 2011, Mary Ann Liebert, Inc.

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FIG. 5. — Impaired retrograde transport following concussive brain trauma. Representative photomicrographs demonstrating Fluoro-Gold (FG)-labeled cell bodies in the cortex. (A) FG-positive cell bodies at the site of injection in sham-injured mice. (B) Note the absence of FG-labeled cell bodies in the cortex contralateral to the site of injection following transection of callosal fibers. (C) FG-positive cell bodies in the homotypic cortex contralateral to the site of injection in sham-injured mice. (D) At 14 days post-injury; note the particulate nature of FG (see also inset in C). (E) Quantification of FG-positive cells was performed as described in the methods section. All values are presented as mean±standard deviation (HPF, high-powered field; *p<0.05 compared to sham-injured animals; scale bar=100 μm for panels A–D, and 10 μm for inset in panel C).

FIG. 5. — Impaired retrograde transport following concussive brain trauma. Representative photomicrographs demonstrating Fluoro-Gold (FG)-labeled cell bodies in the cortex. (A) FG-positive cell bodies at the site of injection in sham-injured mice. (B) Note the absence of FG-labeled cell bodies in the cortex contralateral to the site of injection following transection of callosal fibers. (C) FG-positive cell bodies in the homotypic cortex contralateral to the site of injection in sham-injured mice. (D) At 14 days post-injury; note the particulate nature of FG (see also inset in C). (E) Quantification of FG-positive cells was performed as described in the methods section. All values are presented as mean±standard deviation (HPF, high-powered field; *p<0.05 compared to sham-injured animals; scale bar=100 μm for panels A–D, and 10 μm for inset in panel C).