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. 2011 Apr;28(4):547–563. doi: 10.1089/neu.2010.1729

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Copyright 2011, Mary Ann Liebert, Inc.

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FIG. 7. — Neurodegeneration in the cortex following concussive brain trauma. Representative photomicrographs illustrating Fluoro-Jade-B reactivity in the retrosplenial cortex in (A) sham-injured animals, and (B) at 24 h post-injury. (C) Quantification of the number of Fluoro-Jade-B-positive cells in the cortex below the site of impact. All values are presented as mean±standard deviation (*p<0.05, **p<0.001 compared to sham-injured brains; #p<0.001 compared to 3 or 7 days post-injury). Panels D–F are photomicrographs of Nissl staining in the retrosplenial cortex of (D) sham-injured brains, and at (E) 24 h, and (F) 7 days post-injury. Note the increased cellularity in the brain-injured animals (scale bar=100 μm for all panels).

FIG. 7. — Neurodegeneration in the cortex following concussive brain trauma. Representative photomicrographs illustrating Fluoro-Jade-B reactivity in the retrosplenial cortex in (A) sham-injured animals, and (B) at 24 h post-injury. (C) Quantification of the number of Fluoro-Jade-B-positive cells in the cortex below the site of impact. All values are presented as mean±standard deviation (*p<0.05, **p<0.001 compared to sham-injured brains; #p<0.001 compared to 3 or 7 days post-injury). Panels D–F are photomicrographs of Nissl staining in the retrosplenial cortex of (D) sham-injured brains, and at (E) 24 h, and (F) 7 days post-injury. Note the increased cellularity in the brain-injured animals (scale bar=100 μm for all panels).