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. Author manuscript; available in PMC: 2012 Apr 1.
Published in final edited form as: Dev Dyn. 2011 Feb 28;240(4):808–819. doi: 10.1002/dvdy.22591

Figure 3.

Figure 3

Expression of miR-183 in inner ear of Neurog1 null, Atoh1 null and Pou4f3 mutant mice and controls by in situ hybridization follows neurosensory cell fate specification and differentiation. A: Hair cell expression of miR-183 is unaffected in Neurog1 null inner ear lacking in sensory neurons. B: Expression of miR-183 is evident in remaining sensory neurons of Atoh1 null inner ear lacking hair cells. C: Expression of miR-183 is unaffected in sensory neurons of Pou4f3 mutant inner ear, but absent in vestibular hair cells and weak in remaining cochlear hair cells. Scale bars represent 200 μm. Labels are as described in the legend to Fig. 1.