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. Author manuscript; available in PMC: 2012 May 1.
Published in final edited form as: J Neurochem. 2011 Jan 28;117(3):375–387. doi: 10.1111/j.1471-4159.2011.07175.x

Fig. 6.

Fig. 6

Neither N-acetyl-L-cysteine (NAC) nor vitamin E (VitE) replaced the DJ-1 knock-down-induced deficiency in neuroprotection against rotenone to wild-type levels. MAP2+ (neuronal) signal from bioassay is shown as mean ± SEM (n = 5) after 72 h of treatment with rotenone (Rot, 10 nM in a and c, 20 nM in b and d) ± NAC (a and b) or ± VitE (c and d) at the doses shown. Asterisks (*) represent p < 0.05 vs. same-dose wild-type by paired t-tests.