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. Author manuscript; available in PMC: 2012 Jun 1.

Published in final edited form as: Biotechnol Bioeng. 2011 Feb 24;108(6):1360–1371. doi: 10.1002/bit.23069

Data from engineering the methylmalonate pathway in the absence of propionate, as a function of three strains (BAP1, TB3, and BAB2) and three plasmid systems (a control with only pBP130 and pBP144, an additional pCDFDuet-mce, and pACYCDuet-matBC/pCDFDuet-mce). Panel (a) shows the 6-dEB titer, while panel (b) shows the fraction of methylmalonate utilized as a function of these cellular parameters. Error bars represented ± one standard deviation from three replicates. * indicates statistically significant results (p < 0.05) between the comparisons shown.

Data from engineering the methylmalonate pathway in the absence of propionate, as a function of three strains (BAP1, TB3, and BAB2) and three plasmid systems (a control with only pBP130 and pBP144, an additional pCDFDuet-mce, and pACYCDuet-matBC/pCDFDuet-mce). Panel (a) shows the 6-dEB titer, while panel (b) shows the fraction of methylmalonate utilized as a function of these cellular parameters. Error bars represented ± one standard deviation from three replicates. * indicates statistically significant results (p < 0.05) between the comparisons shown.