Skip to main content
. Author manuscript; available in PMC: 2012 Feb 1.
Published in final edited form as: J Neuroendocrinol. 2011 Feb;23(2):168–176. doi: 10.1111/j.1365-2826.2010.02086.x

Figure 2.

Figure 2

Effect of truncation of the rat DBH promoter on response to E2. (A) Schematic of truncations of DBH promoter. (B) PC12 cells were co-transfected with ERα and β-galactosidase (β-gal) expression vectors and either rDBH(−249/+21)/Luc, rDBH(−200/+21)/Luc, or rDBH(−100/+21)/Luc. Cells were treated with 0 or 10 nM E2 for 24 h and assayed for luciferase activity. Luciferase activity is normalized to β-gal activity and expressed relative to untreated controls. * p≤0.05 compared to respective untreated control; # p≤0.05 compared to rDBH(−200/+21)/Luc treated with E2.