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. Author manuscript; available in PMC: 2011 Apr 14.
Published in final edited form as: Brain Res Bull. 2009 May 27;81(2-3):198–210. doi: 10.1016/j.brainresbull.2009.05.019

Table 1.

Molecules involved in corneal transparency and avascularity.

Corneal crystallins
-aldehyde dehydrogenase (ALDH)
-transketolase (TKT)

Promotor of transformation of keratocyte to corneal myofibroblast
-extracellular matrix metalloproteinase inducer (EMMPRIN)/CD147

Matrix metalloproteinases (MMPs) involved in corneal healing
-Specific expression by corneal fibroblasts; sustained expression
 -MMP-1/fibroblast collagenase
 -MMP-2/gelatinase A
 -MMP-3/stromelysin
-Short-term expression
 -MMP-8/neutrophil collagenase
 -MMP-9/gelatinase B

Signal proteins modulating keratocyte action
-Transforming growth factor beta-1 (TGF-β1)—appears to promote healing/scar formation
-Transforming growth factor beta-3 (TGF- β3)—appears to inhibit healing/scar formation
-Interleukin-10 (IL-10)—appears to inhibit healing/scar formation
-Fibroblast growth factor-2 (FGF-2)—inhibits TGF-β1 expression
-Connective tissue growth factor (CTGF)—synergizes with TGF-β1
-Epidermal growth factor (EGF)—synergizes with TGF-β1

Molecules involved in penetrating keratoplasty graft rejection
-Pro-hemangiogenic/lymphangiogenic
 -Vascular endothelial growth factor (VEGF) isoforms and its associated receptors
   -Vascular endothelial growth factor-A (VEGF-A)—specifically also promotes lymphangiogenesis
   -Vascular endothelial growth factor-C (VEGF-C)—specifically also promotes lymphangiogenesis
   -Vascular endothelial growth factor receptor-3 (VEGF-R3)—specifically also promotes
lymphangiogenesis
 -Cell surface integrin α1β1—presumably involved in endothelial migration
-Anti-angiogenic
 -Fas ligand (FasL)—two forms, mFasL (membrane-bound) and sFasL (soluble); unclear roles of each
 - Thrombospondin 1 (TSP-1)
 - Pigment Epithelial Derived Factor (PEDF)
 - Interferon gamma
 -Soluble VEGF receptors
   -sVEGFR-1 (sFlt-1)
   -sVEGFR-2 (sFlk-1)
-Other signal molecules that promote rejection
 -Tumor necrosis factor alpha (TNF-α)
 -Tumor necrosis factor beta (TNF-β)
 -Interleukin-1 (IL-1)
-Other signal molecules that inhibit rejection
 -Transforming growth factor beta-2 (TGF- β2)