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. Author manuscript; available in PMC: 2011 Sep 10.
Published in final edited form as: Neuroscience. 2010 Dec 31;176:72–85. doi: 10.1016/j.neuroscience.2010.12.041

Figure 6. BDNF-dependent decrease in surface GABAARα1 subunits is partially reversed by PKC inhibition, but not by PKA inhibition in amygdala cultures.

Figure 6

Panel A illustrates light microscopic images of surface (green) GABAARα1 subunits following treatment with vehicle, BDNF, Calphostin C (PKC inhibitor), Rp-8Br-cAMP (PKA inhibitor), or these agents combined with BDNF. Panel B illustrates images of intracellular (red) GABAARα1 subunits following the same conditions as in (A). Panel C demonstrates quantification of fluorescence intensity of surface (black) or intracellular (gray) GABAARα1, normalized with controls. (Mean ± SEM of normalized intensity, * p < 0.05, ** p < 0.01, relative to no treatment.)