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. Author manuscript; available in PMC: 2012 Jun 30.
Published in final edited form as: Respir Physiol Neurobiol. 2011 Mar 21;177(1):47–55. doi: 10.1016/j.resp.2011.03.012

Fig. 2.

Fig. 2

Single-unit carotid chemoafferent nerve responses to (A) 12% O2 and (B) 0% O2 in rats exposed to 21% O2 (Control) or 60% O2 (Hyperoxia) beginning 1 d prior to birth and studied at P1-P7; the shaded region represents the period of hyperoxic exposure. To assess potential recovery of chemoafferent responses, additional Control and Hyperoxia rats were returned to room air at P7 and studied after 3 or 7–8 d recovery (i.e., P10 or P14-15). Values are mean±SEM. Number of observations: Control: P1 (n=12), P2 (n=8), P3 (n=11), P4 (n=9), P5 (n=8), P6 (n= 11), P7 (n=16), P10 (n=11), P14-15 (n=19); Hyperoxia: P1 (n=8), P2 (n=10), P3 (n=10), P4 (n=16), P5 (n=8), P6 (n=6), P7 (n=14), P10 (n=14), P14-15 (n=13). *P<0.05 vs. Control at same age. For comparison, the triangles represent units recorded from P14 rats maintained in 60% O2 from birth (i.e., no normoxic recovery) in an earlier study (Donnelly et al., 2005).