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. 2011 Feb 7;43(4):169–178. doi: 10.3858/emm.2011.43.4.018

Figure 1.

Figure 1

Lung inflammation in the context of spontaneous IFN-γ production is aggravated by the absence of FGF2. (A) The level of secreted FGF2 from fibroblasts (NIH3T3) stimulated by TGF-β1 (10 µg/ml). (B) BAL cellularity of WT, FGF2-deficient mice, non-dox-treated IFN-γ transgenic mice, and IFN-γ transgenic mice crossed with FGF2-deficient mice (n=5). (C) Numbers of CD4+ and CD8+ T cells in the BAL fluid. D. IP-10 and TGF-β1 were assayed in BAL fluid. * and **P <0.05; * denotes a significant difference compared to WT and FGF2-deficient mice and ** denotes a significant difference compared to IFN-γ transgenic mice.