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. 2011 May 5;7(5):e1001127. doi: 10.1371/journal.pcbi.1001127

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Kabaso et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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(a,b) Shapes of adhering cells on a flat two-dimensional surface, with fluorescently labeled actin. We denote lamellipodia by “A” and adhesion domains by “”. (c) A cell dominated by lamellipodia, where we show a segment of the cell perimeter (lower panel) and compare to the calculated (flat geometry) membrane shape (solid line) and cortical actin density (dashed line), assuming a constant rate of actin depolymerization behind the leading edge. (d) Comparison between cell shapes dominated by lamellipodia and the calculated cell shape and cortical actin density (round geometry).

Inline graphic — (a,b) Shapes of adhering cells on a flat two-dimensional surface, with fluorescently labeled actin. We denote lamellipodia by “A” and adhesion domains by “”. (c) A cell dominated by lamellipodia, where we show a segment of the cell perimeter (lower panel) and compare to the calculated (flat geometry) membrane shape (solid line) and cortical actin density (dashed line), assuming a constant rate of actin depolymerization behind the leading edge. (d) Comparison between cell shapes dominated by lamellipodia and the calculated cell shape and cortical actin density (round geometry).