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A unified theory of cancer immune complexity. Tumours comprised of a heterogenous population of cells that expands from clones that send danger signals to the immune system (red) to clones that do not appear to be dangerous to the immune system (blue). The balance between these two clones determines the state of tumour immunosurveillance in the host. There are 10 possible pathways depicted here, which describes the interplay between tumour immunoediting and tolerance.
