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. Author manuscript; available in PMC: 2012 Jun 1.
Published in final edited form as: Aging Cell. 2011 Mar 31;10(3):429–437. doi: 10.1111/j.1474-9726.2011.00679.x

Figure 2. Superoxide-, NADPH oxidase- and tetrahydrobiopterin-dependent modulation of endothelial-dependent dilation.

Figure 2

(A) Maximal dilation of carotid arteries to acetylcholine (ACh) and ACh + TEMPOL, a superoxide dismutase mimetic. (B) Maximal dilation of carotid arteries to ACh and to ACh + apocynin, a NADPH oxidase inhibitor. (C) Maximal dilation of carotid arteries to ACh and to ACh + sepiapterin, an exogenous tetrahydrobiopterin donor. Values are mean ± SEM. (n = 6 – 9 per group) * p < 0.05 vs. YC.