Skip to main content
. Author manuscript; available in PMC: 2012 May 1.
Published in final edited form as: Pediatr Res. 2011 May;69(5 Pt 1):406–412. doi: 10.1203/PDR.0b013e3182110c7d

Figure 6.

Figure 6

At study completion, male and female GR mice weighed significantly less than their control counterparts (A; males: F(1,28)=7.7, P=0.01; females: F(1,27)=4.9, P<0.05), but post-hoc testing did not isolate significant differences within subgroups of saline-treated or leptin-treated mice (■: control-saline, n=7 male, 8 female; □: GR-saline, n=9 male, 8 female; ▨: control-leptin, n=7 male, 9 female; ▤: GR-leptin, n=9 male, 6 female ). Neonatal GR deceased adult male length (B; F(1,18)=4.3, *P<0.05 versus control-saline). Furthermore, neonatal leptin administration increased the length of adult male mice (B, F(1,18)=7.9, †P<0.01 versus GR-saline). In female mice, neonatal GR increased relative brain weights (C, F(1,27)=8.1, *P<0.05 versus control-leptin). In male mice, neonatal GR decreased white adipose tissue (D, F(1,28)=7.6, *P<0.05 GR-saline versus control-saline and P=0.18 GR-leptin versus control-leptin) and brown adipose tissue (E, F(1,28)=6.0, P=0.28 GR-saline versus control-saline and *P<0.05 GR-leptin versus control-leptin).