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. 2011 Apr 14;8:34. doi: 10.1186/1742-2094-8-34

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Copyright ©2011 Ramberg et al; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Increased levels of C/EBPβ in brain areas with high Aβ plaque burdens from aged tg-ArcSwe mice. A) Relative abundance of C/EBPβ showing increased protein levels in forebrain of aged tg-ArcSwe mice (tg) compared to non-transgenic mice (c). B) Representative western blot showing two isoforms of C/EBPβ (LAP and LIP). Only the C/EBPβ LAP isoform was quantified. The data represent mean ± SEM for six transgenic and six control mice. 40 μg of protein lysate was loaded in each lane and β-actin was used as a loading control. *p < 0.05, significantly different from control; crm, cross-reactive material (cf., [53,54]). C/EBPβ protein levels in hindbrain from aged mice and from both forebrain and hindbrain from young mice were below or close to detection limit. A.U., arbitrary units.