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Inhibition of cPKCβII had no significant effect on smooth muscle function. A, neither FK506 nor treatment with the cPKCβI or cPKCβII isoform-specific peptide inhibitors affected aortic endothelium-independent relaxation responses. B, the cPKCγ isoform-specific peptide inhibitor or inhibition of all cPKC isoforms had no effect on relaxation responses to the endothelium-dependent dilator sodium nitroprusside. *, p < 0.05. The numbers of aortas are given in parentheses.
