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. Author manuscript; available in PMC: 2012 May 1.
Published in final edited form as: Mol Microbiol. 2011 Apr 11;80(4):1014–1030. doi: 10.1111/j.1365-2958.2011.07627.x

Fig. 3.

Fig. 3

Effects of sinR, abrB and clpC inactivation on eps-lacZ expression in wild-type (BD4498) and pKD93 (BD4643) backgrounds. Strains carrying the indicated mutations, were grown in LB and samples taken for β-galactosidase determination at T1. (A) The following strains, all carrying eps-lacZ, were used for this experiment: BD4498 (wild-type), BD4644 (pKD93), BD4544 (sinR::cat), BD4549 (sinR::cat pKD93), BD4623 (abrB::cat), BD4615 (sinR::kan abrB::cat), BD5113 (abrB::cat pKD93), BD4624 (abrB::cat sinR::kan pKD93). (B) The following strains carrying eps-lacZ were used for this experiment: wild-type (BD4498), sinR::cat (BD4544), abrB::cat (BD4623), spo0A::kan (BD4928), spo0A::kan sinR::cat (BD4929), spo0A::kan abrB::cat eps-lacZ (BD4930), spo0A::kan abrB::cat sinR::kan (BD4931), spo0A::kan mecA::erm (BD5589), mecA::erm (BD4538). (C) The following strains were used for this experiment: wild-type (BD4498), pKD93 (BD4644), clpC::tet (BD4580), clpC::tet pKD93 (BD5114). The presence of the pKD93 plasmid indicates that mecA is over-expressed. The numbers in parentheses refer to the number of independent measurements for each strain and the whiskers show standard deviations.