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Mechanism of pulmonary dilatation in response to some specific drugs and Rho-kinase inhibitors. 5-HT, serotonin; CaM, calmodulin; CCBs, calcium channel blockers; ET-1, endothelin-1; MLCK, myosin light chain kinase; MLCPh, myosin light chain phosphatase; NO, nitric oxide; PDE-5, phosphodiesterase-5; VSMC, vascular smooth muscle cell; PGI2, prostacyclin; PLC, phospholipase C; SR, sarcoplasmic reticulum. Reproduced with permission from Fukumoto et al. (2007).
