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. Author manuscript; available in PMC: 2011 Jun 6.

Published in final edited form as: Autoimmunity. 2008 Sep;41(6):442–453. doi: 10.1080/08916930802031579

(A) Comparison of sera from myocarditis, dilated cardiomyopathy, giant cell myocarditis and normal control patients in their reactivity with 82 synthetic peptides spanning the S2 (25-mers) hinge region and LMM (18-mers) tail region of the human cardiac myosin rod in the ELISA (sera @ 1/100 dilution). Significant reactivity (OD) compared to normal controls for the peptides is indicated with p values and grey bars. (B) Comparison diabetic, diabetic cardiomyopathy, and normal sera reactivity with S2 and LMM synthetic peptides. Sera from the control donors rarely reacted with S2 peptides at an OD >0.2. Significant reactivity compared to normal controls is indicated by p values and grey bars.

(A) Comparison of sera from myocarditis, dilated cardiomyopathy, giant cell myocarditis and normal control patients in their reactivity with 82 synthetic peptides spanning the S2 (25-mers) hinge region and LMM (18-mers) tail region of the human cardiac myosin rod in the ELISA (sera @ 1/100 dilution). Significant reactivity (OD) compared to normal controls for the peptides is indicated with p values and grey bars. (B) Comparison diabetic, diabetic cardiomyopathy, and normal sera reactivity with S2 and LMM synthetic peptides. Sera from the control donors rarely reacted with S2 peptides at an OD >0.2. Significant reactivity compared to normal controls is indicated by p values and grey bars.