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Propranolol, a beta-adrenergic receptor antagonist, is shown to effectively inhibit the PKA activity of the purified IgG from selected cardiomyopathy sera. Purified IgG from cardiomyopathy activated PKA significantly above basal PKA levels while purified normal IgG did not. H9c2 heart cells in culture were treated with purified IgG and PKA activity measured in counts per minute as described in materials and methods.
